NEUROLOGY

Motor neurone disease

  • Motor system failure caused by progressive degeneration of UMNs (corticospinal and corticobulbar tracts) and LMNs (anterior horn cells)
  • presents with upper or lower motor neurone signs, or a mixture of the two
  • Progressive weakness of limbs, bulbar and respiratory muscles
  • Does not involve sensory system
  • incidence: approximately 2 per 100 000 per year except in endemic areas such as the Island of Guam
  • prevalence: 5-6 per 100 000 of the population
  • males affected more than females in ratio of 2:1
  • familial link in 5-10% of cases
  • age of onset – mean = 55 years; range = 16-77 years, but usually in fifth to seventh decades
  • 3 subtypes
    • Amyotrophic lateral sclerosis – ALS – most common – UMN and LMN signs, a late onset, rapidly progressive and ultimately fatal neurological disorder
    • Progressive muscular atrophy – LMN features only
    • Progressive bulbar – LMN in brainstem motor nuclei 
    • Primary lateral sclerosis – UMN signs only
  • Clinical
    • most patients visit their general practitioner first, typically with mild symptoms such as 
      • Cramps
      • balance disturbance
      • reduced dexterity
      • Subtle cognitive changes including apathy
      • time from symptom onset to diagnosis ranges from 10 – 16 months
      • signs often go unrecognized, with patients referred to other specialists, or given misdiagnoses
    • Asymmetrical weakness in upper or lower limbs
    • Weakness
    • Mixed UMN / LMN signs
      • brisk reflexes in a limb with muscle wasting
    • Absence of sensory symptoms/pain
    • Steadily progressive weakness
    • Widespread fasiculations  
    • Preferential wasting lateral hand border
    • Extra motor symptoms 50%
      • cognitive and behavioural impairment
      • language abnormalities
    • Can have fronto-temporal dementiam – similar mechanisms

  • Investigations
    • If suspected needs referral to neurologist
    • To exclude other causes MRI brain/spine
    • Bloods including autoimmune screen, anti-ganglioside antibodies, ACH receptor antibodies, toxins, heavy metals, HIV, HIV , inflamamtory disease
    • May get nerve conduction studies or EMG
  • Counselling
    • Incurable, usually death 3-5 years
    • 90% have no family history, Often don’t find the gene
    • Risk to first degree relatives 1-3% if not known to be familial
  • Treatment
    • Multi-disciplinary care
    • Riluzole – protective for disease progression, survival benefit 3-6 months – monitor LFTs and neutropaenia monthly
    • Early respiratory support – consider nocturnal NIV
    • Maintain weight

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