DERMATOLOGY,  HAIR

Alopecia areata

Alopecia areata is an autoimmune condition causing hair loss, typically presenting as discrete bald patches on the scalp and potentially affecting all hair-bearing areas.

Variants of Alopecia Areata

  • Alopecia Totalis: Complete scalp hair loss.
  • Alopecia Universalis: Total body hair loss.
  • Ophiasis: Bandlike pattern of hair loss.
  • Ophiasis Inversus: Reverse bandlike pattern.
  • Diffuse Alopecia Areata: Widespread thinning.

Prevalence and Demographics

  • Lifetime Risk: About 2%.
  • Affected Groups: Children and adults of all skin/hair colors, with peak incidence in the second and third decades of life.

Risk Factors

  • Chromosomal disorders (e.g., Down syndrome).
  • Polyglandular autoimmune syndrome type 1.
  • Other autoimmune conditions (e.g., vitiligo, thyroid disease).
  • Family history of alopecia areata.
  • Specific susceptibility genes.

Pathogenesis

  • Involves loss of immune privilege in anagen hair follicles, leading to autoimmune attack.
  • Supported by perifollicular T cell infiltrates observed in histopathology.
  • Strong genetic component with at least 16 risk loci identified.

Clinical Presentation

  • Patterns of Hair Loss:
    • Most common: Patchy alopecia areata.
    • Others: Alopecia totalis, universalis, ophiasis, sisaipho, and diffuse alopecia areata.
  • Features: Exclamation point hairs, localized tingling/itching, potential regrowth of unpigmented hairs.
  • Nail Changes: Pitting, ridging in 10–40% of patients.

Diagnosis

  • Primary Diagnosis: Clinical.
  • Diagnostic Tools:
    • Trichoscopy may reveal specific features (e.g., exclamation point hairs, yellow dots).
    • Hair pull test and skin biopsy can aid diagnosis.

Differential Diagnosis

  • Trichotillomania.
  • Tinea capitis.
  • Telogen effluvium.
  • Androgenetic alopecia.
  • Discoid lupus erythematosus.

Treatment

  • No Cure: Treatment aims to speed up hair regrowth or camouflage hair loss.
  • Mild Cases: Intralesional corticosteroids, topical treatments (corticosteroids, minoxidil, anthralin).
  • Extensive Cases: Topical immunotherapy, systemic corticosteroids, JAK inhibitors.
  • Other Treatments: Dupilumab, methotrexate, platelet-rich plasma, micro-needling.
  • Camouflage Options: Wigs, hairpieces, and styling products.

Prevention

  • Currently, no known preventive measures for alopecia areata.

Prognosis

  • Course: Unpredictable; spontaneous regrowth common but relapses possible.
  • Progression:
    • 5–10% of cases progress to totalis or universalis with low likelihood of recovery.
  • Poor Prognosis Factors: Early onset, extensive disease, nail dystrophy, ophiasis pattern, comorbid autoimmune diseases.

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